A Triad of Interacting Mechanisms Driving the Pathophysiology of AD, Asthma, CRSwNP, and EoE

• Discover how systemic and local type 2 inflammation contribute to the pathophysiology of atopic dermatitis (AD), asthma, chronic rhinosinusitis with nasal polyps (CRSwNP), and eosinophilic esophagitis (EoE)
• Delve into how barrier dysfunction, neuroimmune dysregulation, and type 2 inflammation are associated with signs and symptoms of AD, asthma, CRSwNP, and EoE
• Briefly examine how microbial dysbiosis contributes to the pathophysiology of AD, asthma, CRSwNP, and EoE

This infographic delves into the role of barrier dysfunction, neuroimmune dysregulation, and type 2 inflammation in the pathophysiology of dermatological, respiratory, and gastroenterological diseases. Discover how a triad of aberrant interacting mechanisms drive recurring and debilitating symptoms in a range of diseases with underlying type 2 inflammation.