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Dermatology

A Driver of Pruritic Dermatoses: Type 2 Inflammation is Central to Skin Barrier and Neuroimmune Dysregulations

Uncover the role of type 2 inflammation in pruritic dermatoses, including barrier disruption and the neuroimmune axis – an ADVENT webinar from 2022.

Sonja Ständer
Lisa Beck
MD
Sonja Ständer +1 more

Learning objectives

  • Review the latest understanding and clinical importance of the triad of interrelated mechanisms that drive atopic dermatitis
  • Explore the immunological and pathophysiological implications of S. aureus colonization of the skin in atopic dermatitis
  • Explore innovative methods to evaluate skin barrier function and data on therapeutic interventions to normalize barrier function in atopic dermatitis

Description

In this ADVENT medical education webinar from 2022, Prof. Lisa Beck, MD (University of Rochester, USA), and Prof. Sonja Ständer, MD (University of Münster), discuss type 2 inflammation as a key driver of pruritic dermatoses. Additionally, they emphasize the significance of type 2 inflammation in compromising the skin barrier, its link to infections, and its influence on the neuroimmune axis.

Tags
disease backgroundexpert insightsmechanism of disease
MAT-GLB-2204070 V2 09/2022

About these experts

Dermatology
MD

Sonja Ständer

Professor of Dermatology and Neurodermatology at the University of Münster; Head of the Interdisciplinary Center for Chronic Pruritus (KCP) at the University Hospital Münster, Germany

Dermatology
MD

Lisa Beck

Goldsmith Professor of Dermatology, University of Rochester Medical Center, New York, United States

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Sanofi and Regeneron are global leaders in pharmaceutical development, each with a long history of supporting frontline physicians to improve human health and reduce the burden of disease. Driven by the same commitment to patients and their families, Sanofi and Regeneron have partnered to take on type 2 inflammation and the range of chronic conditions in which excessive inflammation plays a role.

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