110 search results for: burden of disease

ADVENT On Air | Loss <b>of</b> Smell in CRSwNP Patients
Rhinology
ADVENT On Air | Loss of Smell in CRSwNP Patients
Podcast

Profs. Wytske Fokkens and Joaquim Mullol discuss the clinical significance of the loss of smell in patients with CRSwNP.

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Interplay <b>of</b> Barrier Dysfunction and Type 2 Inflammation in EoE
Gastroenterology
Interplay of Barrier Dysfunction and Type 2 Inflammation in EoE
expert video

Learn from Prof. Seema Aceves about the interconnectivity of type 2 inflammation and barrier dysfunction in mediating EoE pathophysiology.

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ERS 2026 | Distal Airways, Central Role? Clinical Insights Into Type 2 Severe Asthma
Pulmonology
ERS 2026 | Distal Airways, Central Role? Clinical Insights Into Type 2 Severe Asthma
expert video

Join experts Frederik Trinkmann, Monica Kraft, and Arnaud Bourdin on an ADVENT educational symposium, “Distal Airways, Central Role? Clinical Insights Into Type 2 Severe Asthma."

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Complex EoE Pathophysiology Beyond Esophageal Eosinophilia
Gastroenterology
Complex EoE Pathophysiology Beyond Esophageal Eosinophilia
expert video

Dr. Jonathan Spergel and Prof. Arjan Bredenoord highlight the importance of different immune cells in type 2 inflammation in EoE.

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ERS-ISIAN 2025 | Treatable Traits in CRSwNP
Rhinology
ERS-ISIAN 2025 | Treatable Traits in CRSwNP
expert video

Join Prof. Sietze Reitsma for an insightful presentation that will delve into identifying and leveraging key characteristics to optimize patient management and treatment outcomes.

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CRSwNP Interactive Patient Case Tool
Rhinology
CRSwNP Interactive Patient Case Tool
interactivity

Meet Fernando. Journey through his clinical profile, applying guideline-based checklists to reach a consensus regarding his diagnosis.

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More Than Eosinophils: A Closer Look at the Pathophysiology <b>of</b> EoE
Gastroenterology
More Than Eosinophils: A Closer Look at the Pathophysiology of EoE
expert video

Join Prof. Kathryn Peterson for an expert review on how different immune cells and inflammatory mediators contribute to the pathogenesis of EoE.

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What Went Wrong? How Dysregulated Type 2 Immunity Contributes to AD, PN, CSU, and BP
On Demand
What Went Wrong? How Dysregulated Type 2 Immunity Contributes to AD, PN, CSU, and BP

The March 2025 ADVENT symposium in Orlando, Florida brought together 4 dermatology experts to explore the evolving science of type 2 inflammation. Type 2 inflammation plays a central role in the pathophysiology of multiple dermatological diseases, driving chronic immune dysregulation that affects patients with atopic dermatitis (AD) prurigo nodularis (PN), chronic spontaneous urticaria (CSU) and bullous pemphigoid (BP). Understanding the mechanisms behind type 2 inflammation is key to advancing care and improving patient quality of life.

Spotlight on Type 2 Inflammation in the Upper and Lower Airways
Pulmonology
Spotlight on Type 2 Inflammation in the Upper and Lower Airways
expert video

In this video soundbite from the EAACI 2025 symposium, Dr. Zuzana Diamant explains pathophysiological mechanisms underlying asthma and CRSwNP, and how type 2 inflammatory cytokines drive airway remodelling in patients with severe asthma. Additionally, patients with co-existing asthma and CRSwNP have a higher type 2 inflammatory burden than patients with asthma alone.

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Master Clinician Video | Advanced Clinical Practice in EoE (Full)
Gastroenterology
Master Clinician Video | Advanced Clinical Practice in EoE (Full)
videoanimation

Learn how a food bolus impaction event can help identify a patient with EoE and the importance of multiple disease domains in assessing EoE.

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ADVENT Forum 2025 On Demand page
On Demand
ADVENT Forum 2025 On Demand page

Watch on-demand expert soundbites recorded at the April 2025 ADVENT Forum in Lisbon, Portugal, featuring dermatology and immunology perspectives on mechanisms and clinical practice. Topics range from disease mechanism to patient care and quality of life, spanning atopic dermatitis (AD), prurigo nodularis (PN), chronic spontaneous urticaria (CSU) and bullous pemphigoid (BP).